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Tuesday, November 15, 2016

Arts + Sciences Events

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Physics SPS Seminar: "Physics in Neuroscience and Studying Central Congenital Deafness"

Time: 12:30 pm to 1:30 pm

Contact: Chelsea Fielding · cafieldi@loyno.edu · 504-865-3647

Location: Monroe Hall, Rm 152

"A Primer on the Physics in Neuroscience and Studying Central Congenital Deafness"

       Speaker: Dave Vumbaco, The Neuroscience Center of Excellence, LSUHSC

 

The seminar will be given in two parts. In the first part we will discuss the connections between physics and neuroscience. The abstract for the second part is as follows:

 

   Usher syndrome (Usher) is the most common genetic disease causing combined deafness and blindness. The c.216G>A mutation (216A) in the USH1C gene, which is responsible for type 1 Usher in Acadian populations, causes aberrant splicing that results in a frameshift and a truncated RNA and Harmonin protein. Mice homozygous for the 216A mutation (Ush1c216AA) are deaf, and have vestibular and visual dysfunctions, similar to patients. Treatment of Ush1c216AA mice with antisense oligonucleotides (ASOs) rescues hearing, balance, and vision. The organization of the central auditory structures associated with Usher is unknown. We hypothesize that there are anatomical changes in central auditory structures including the primary auditory cortex (A1) and the ventral medial geniculate nucleus (MGv) in Usher mice. To explore this hypothesis, Ush1c216AA and normal hearing littermates were injected with BDA conjugated to a red fluorophore (BDA-red) into the MGv, and in the right primary auditory cortex with BDA conjugated to a green fluorophore (BDA-green). The intersection of the neural projections from the right A1 and the left MGv into left A1 was quantified along the mediolateral and rostrocaudal axes. Preliminary results suggest that the neuroanatomical pathways are significantly different in Ush1c216AA mice compared with normal hearing littermates. The quantity of BDA-red fluorescence is greater in Ush1c216AA mice compared to littermate controls, suggesting that the MGv projection into ipsilateral left A1 is expanded. Quantification of the BDA-green shows a reduction in fluorescence in the rostral region of A1 and an increase in the caudal region of A1 compared to littermate controls, suggesting the disorganized commissural right A1 to left A1 projections. Taken together, these results show, for the first time, defects in the central ascending auditory pathway in addition to peripheral defects associated with Usher syndrome.

 

Pizza & Drinks will be available.

See you there!

Networking

Time: 5:30 pm to 7:30 pm

Contact: Laura Jayne · ljayne@loyno.edu · 5048653431

Location: Communications/Music Complex Room 401

Business professionals from the New Orleans community come to Loyola to recruit and network with students. All students are invited.

Tomb Raiders & Terrorist Financing: The Islamic State's Illicit Traffic in 'Blood Antiquities'

Time: 8 pm to 9 pm

Contact: Dr. Connie Rodriguez · rodrigue@loyno.edu · 504.865.2287

Location: Whitney Bank Presentation Room, Thomas Hall

A Lecture by

Tess Davis

Executive Director

Antiquities Coalition

free admission and free parking on campus (West Road Garage and the Horseshoe)

Co-sponsored by the Office of the Provost, the Department of Classical Studies and the New Orleans Society of the Archaeological Institute of America

With the rise of the Islamic State in Iraq and Syria (ISIS), the world rightfully asked how a militant faction too extreme for Al-Qaeda transformed itself into “the world’s richest terror group ever.” ISIS boasts an annual budget worth $2 billion and a war chest of $250 million, which if true surpasses the Taliban’s (and that of many states). Still more troubling, it is now financially self sufficient, and no longer dependent on foreign donors. 

How? Like organized criminal enterprises before it: extortion, ransom, robbery, and smuggling. It perhaps comes as no shock that it has been trafficking arms, drugs, and even oil. However, the public reacted with surprise to reports in June 2014 that ISIS jihadists had earned “millions” by looting the region’s archaeological sites, and then selling its ancient treasures to the highest bidder. 

It shouldn’t have. Archaeologists, criminologists, law enforcement agents, and military officials have long warned that the illicit antiquities trade is funding crime and conflict around the world. However, under ISIS’ black flag, this looting and trafficking has become not just a side enterprise, but a massive illegal industry. 

In recent years, we have lost some of the Cradle of Civilization's most iconic masterpieces and sites, many of which had survived for millennia. This wanton destruction is erasing our shared history chapter by chapter. And it threatens us all: at this moment, ISIS is converting these "blood antiquities" into weapons and troops, which are seizing cities, slaughtering soldiers, and beheading civilians. 

 

Tess Davis, a lawyer who has dedicated more than a decade to combatting the illicit antiquities trade, will examine this growing threat to our national security and the world's cultural heritage. In doing so, she will trace the past of looted masterpieces from conflict zones to the very height of the global market, and explore how United States and international law is seeking to cut off this key means of criminal financing. Finally, she will discuss recent progress in this fight, as an unprecedented coalition of countries have joined forces to demand action from both governments and the art market.